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Hamamatsu ccd camera (hisca; c6790
Ccd Camera (Hisca; C6790, supplied by Hamamatsu, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ccd+camera+(hisca%3B+c6790/ccd+camera+c6790/pmc08615619-97-40-45
Average 90 stars, based on 1 article reviews
ccd camera (hisca; c6790 - by Bioz Stars, 2026-10
90/100 stars

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Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts.
Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts.

Article Title: Dual effect of capsaicin on cell death in human osteosarcoma G292 cells.
Article Snippet: Thirty percent of osteosarcoma patients die within 5 years.. New agents that induce apoptosis of osteosarcoma cells might be therapeutically useful.. Here, we characterized the apoptotic mechanism induced by capsaicin in G292 osteosarcoma cells.

Article Title: Spectral response properties of higher visual neurons in Drosophila melanogaster.
Article Snippet: Abstract The fruit fly Drosophila melanogaster can process chromatic information for true color vision and spectral preference.. Spectral information is initially detected by a few distinct photoreceptor channels with different spectral sensitivities and is processed through the visual circuit.. The neuroanatomical bases of the circuit are emerging.

Article Title: Generation of Functional Cardiomyocytes from Human Gastric Fibroblast-Derived Induced Pluripotent Stem Cells
Article Snippet: The coverslips were mounted in a modified Cunningham chamber attached to the stage of a fluorescence microscope (model DMIRB; Leica, Mannheim, Germany) that was equipped with a high-speed scanning polychromatic light source (model C7773; Hamamatsu Photonics, Shizuoka, Japan) and a CCD camera (HISCA; model C6790; Hamamatsu Photonics) controlled by Aquacosmos 2.5 software (Hamamatsu Photonics).

Article Title: Inhibition of protein kinase C promotes differentiation of neuroblastoma × glioma NG108-15 hybrid cells.
Article Snippet: Differentiation of neuroblastoma · glioma NG108-15 hybrid cells can be induced by different means, but the mechanisms involved are unclear.. Our aim was to characterize the role of protein kinase C (PKC) in this process.. The PKCs present in NG108-15 cells, i.e. PKCa, PKCd, PKCe and PKCf, were inhibited using a cocktail of Go6983 and Ro318220 or were downregulated by treatment with phorbol 12-myristate 13-acetate (PMA).

Fluorescence:

Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells.
Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway.

Microscopy:

Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells.
Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway.

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Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells.
Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway.



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